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Mechanistically, EMPA and TRF modulated Dox-induced inflammation in distinct ways: both (alone or combined) lowered TNF, TRF alone produced the strongest IL-1 suppression, the combined treatment increased IL-10 and TGF- significantly

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Moreover, all the AcM inclusion levels led to a decreased abundance of KOs encoding for the biosynthesis/metabolism of structural cell components, such as teichoic acid ( tagA , tagE , dltA , dltB , dltC , dltD , cpoA , mgs and bcrC ) and peptidoglycan ( murA , murB , murC , murD , murE , murF , murG , bcrC , dacC , ddl , mraY , pbp2A and pbpB ) of which murF , murG , mraY and ddl together with alr also led to a decrease in intrinsic vancomycin resistance